Immunity to Asexual Blood Stages of Plasmodium : Is Resistance to Acute Malaria Adaptive or Innate?
Identifieur interne : 002755 ( Main/Exploration ); précédent : 002754; suivant : 002756Immunity to Asexual Blood Stages of Plasmodium : Is Resistance to Acute Malaria Adaptive or Innate?
Auteurs : A. H Fell [États-Unis, Royaume-Uni] ; N. C Smith [Australie]Source :
- Parasitology Today [ 0169-4758 ] ; 1998.
English descriptors
- Teeft :
- Acute malaria, Acute parasitaemia, Adoptive transfer, Anopheles, Anopheles dirus, Anopheles gambiae, Antibody production, Antimalarial antibodies, Asexual, Asexual blood stages, Blood cells, Bloodstage, Bloodstage malaria, Chabaudi, Chabaudi adami, Chain knockout mice, Clone, Cytokine, Different circumstances, Elsevier science, Falciparum, High levels, Human malaria, Immune, Immune adults, Immune response, Immune responses, Immunity, Immunol, Infection, Initial parasitaemia, Initial peak, Innate immunity, Intact mice, Kamini mendis, Knockout, Knockout mice, Macrophage, Macrophage activation, Major role, Malaria, Malaria immunity, Malaria infection, Malaria infections, Malaria parasite plasmodium falciparum, Malaria transmission, Mouse, Murine, Murine models, Naive mice, Nitric oxide, Nude mice, Other cytokines, Oxford university press, Parasitaemia, Parasite, Parasite growth, Parasite immunol, Parasite virulence, Parasitol, Parasitology, Parasitology today, Peak parasitaemia, Plasmodium, Plasmodium chabaudi chabaudi, Plasmodium falciparum, Plasmodium falciparum clones, Plasmodium falciparum merozoite surface protein, Plasmodium vivax, Plasmodium yoelii, Protective immunity, Receptor, Richard carter, Rodent malaria, Scid mice, Similar kinetics, Southern asia, Susceptible mice, Tropical africa, Yoelii.
Abstract
Abstract: Current models of immunity to blood stages of Plasmodium invoke a primary role for T-cell dependent processes and much recent evidence implicates Th1-type responses as crucial to the control of acute malaria. But do these data stand up to close scrutiny? Here, Andy Fell and Nick Smith review recent data from rodent and human studies and suggest that Th1-type responses may not after all be important in controlling malaria infection in the blood.
Url:
DOI: 10.1016/S0169-4758(98)01298-8
Affiliations:
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